Pathology
Metabolic acidosis
Metabolic alkalosis
Respiratory acidosis
Respiratory alkalosis
Acute tubular necrosis
Postrenal azotemia
Prerenal azotemia
Renal azotemia
Horseshoe kidney
Potter sequence
Renal agenesis
Hypercalcemia
Hyperkalemia
Hypermagnesemia
Hypernatremia
Hyperphosphatemia
Hypocalcemia
Hypokalemia
Hypomagnesemia
Hyponatremia
Hypophosphatemia
Hydronephrosis
Kidney stones
Renal cortical necrosis
Renal papillary necrosis
Alport syndrome
Goodpasture syndrome
IgA nephropathy (NORD)
Lupus nephritis
Poststreptococcal glomerulonephritis
Rapidly progressive glomerulonephritis
Amyloidosis
Diabetic nephropathy
Focal segmental glomerulosclerosis (NORD)
Lupus nephritis
Membranoproliferative glomerulonephritis
Membranous nephropathy
Minimal change disease
Acute pyelonephritis
Chronic pyelonephritis
Medullary cystic kidney disease
Medullary sponge kidney
Multicystic dysplastic kidney
Polycystic kidney disease
Chronic kidney disease
Renal tubular acidosis
Angiomyolipoma
Beckwith-Wiedemann syndrome
Nephroblastoma (Wilms tumor)
Renal cell carcinoma
WAGR syndrome
Renal artery stenosis
Acid-base disturbances: Pathology review
Congenital renal disorders: Pathology review
Electrolyte disturbances: Pathology review
Kidney stones: Pathology review
Nephritic syndromes: Pathology review
Nephrotic syndromes: Pathology review
Renal and urinary tract masses: Pathology review
Renal failure: Pathology review
Renal tubular acidosis: Pathology review
Renal tubular defects: Pathology review
Urinary incontinence: Pathology review
Urinary tract infections: Pathology review
Acute tubular necrosis
0 / 17 complete
0 / 2 complete
of complete
of complete
Laboratory value | Result | ||
Urinalysis | |||
On admission | Day 2 | Day 8 | |
Creatinine | 1.1 mg/dL | 3.5 mg/dL | 2.3 mg/dL |
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acute tubular necrosis p. 627
acute tubular necrosis p. 627
acute tubular necrosis p. 627
acute tubular necrosis with p. 626
acute tubular necrosis and p. 627
acute tubular necrosis p. 627
acute tubular necrosis with p. 627
acute tubular necrosis from p. 626
acute tubular necrosis p. 626
acute tubular necrosis p. 627
acute tubular necrosis p. 627
acute tubular necrosis p. 627
Acute tubular necrosis (ATN) is a type of acute kidney injury (AKI) that results in the sudden and rapid death of tubular cells in the kidneys. This can lead to a decrease in urine production and an increase in the levels of creatinine and urea in the blood.
ATN may be caused by sepsis leading to hypotension and renal hypoperfusion; ischemia due to renal hypoperfusion, like in hypovolemic shock; but it can also be due to exposure to toxins and nephrotoxic drugs which cause damage to the tubular cells. Such drugs include nephrotoxic antibiotics, contrast agents used during imaging studies, amphotericin B, and toxic heavy metals.
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