Acute tubular necrosis
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Acute tubular necrosis
Pathology
Renal and ureteral disorders
Renal agenesis
Horseshoe kidney
Potter sequence
Hyperphosphatemia
Hypophosphatemia
Hypernatremia
Hyponatremia
Hypermagnesemia
Hypomagnesemia
Hyperkalemia
Hypokalemia
Hypercalcemia
Hypocalcemia
Renal tubular acidosis
Minimal change disease
Diabetic nephropathy
Focal segmental glomerulosclerosis (NORD)
Amyloidosis
Membranous nephropathy
Lupus nephritis
Membranoproliferative glomerulonephritis
Poststreptococcal glomerulonephritis
Goodpasture syndrome
Rapidly progressive glomerulonephritis
IgA nephropathy (NORD)
Lupus nephritis
Alport syndrome
Kidney stones
Hydronephrosis
Acute pyelonephritis
Chronic pyelonephritis
Prerenal azotemia
Renal azotemia
Acute tubular necrosis
Postrenal azotemia
Renal papillary necrosis
Renal cortical necrosis
Chronic kidney disease
Polycystic kidney disease
Multicystic dysplastic kidney
Medullary cystic kidney disease
Medullary sponge kidney
Renal artery stenosis
Renal cell carcinoma
Angiomyolipoma
Nephroblastoma (Wilms tumor)
WAGR syndrome
Beckwith-Wiedemann syndrome
Bladder and urethral disorders
Posterior urethral valves
Hypospadias and epispadias
Vesicoureteral reflux
Bladder exstrophy
Urinary incontinence
Neurogenic bladder
Lower urinary tract infection
Transitional cell carcinoma
Non-urothelial bladder cancers
Renal system pathology review
Congenital renal disorders: Pathology review
Renal tubular defects: Pathology review
Renal tubular acidosis: Pathology review
Acid-base disturbances: Pathology review
Electrolyte disturbances: Pathology review
Renal failure: Pathology review
Nephrotic syndromes: Pathology review
Nephritic syndromes: Pathology review
Urinary incontinence: Pathology review
Urinary tract infections: Pathology review
Kidney stones: Pathology review
Renal and urinary tract masses: Pathology review
Assessments
Acute tubular necrosis
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USMLE® Step 1 questions
0 / 2 complete
High Yield Notes
10 pages
Flashcards
Acute tubular necrosis
of complete
Questions
USMLE® Step 1 style questions USMLE
of complete
| Laboratory value | Result | ||
| Urinalysis | |||
| On admission | Day 2 | Day 8 | |
| Creatinine | 1.1 mg/dL | 3.5 mg/dL | 2.3 mg/dL |
Memory Anchors and Partner Content
External References
First Aid
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Acute tubular necrosis p. 626
Aminoglycosides p. 188
acute tubular necrosis p. 627
Cisplatin p. 449
acute tubular necrosis p. 627
Granular casts p. 618
acute tubular necrosis p. 627
Heart failure p. 318
acute tubular necrosis with p. 626
Hemoglobinuria
acute tubular necrosis and p. 627
Hemorrhage
acute tubular necrosis p. 627
Hypotension
acute tubular necrosis with p. 627
Ischemia p. 208, 691
acute tubular necrosis from p. 626
Lead poisoning p. 427, 432
acute tubular necrosis p. 626
Myoglobinuria
acute tubular necrosis p. 627
Sepsis
acute tubular necrosis p. 627
Shock p. 319
acute tubular necrosis p. 627
Tubular necrosis p. 618, 626, 626
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Summary
Acute tubular necrosis (ATN) is a type of acute kidney injury (AKI) that results in the sudden and rapid death of tubular cells in the kidneys. This can lead to a decrease in urine production and an increase in the levels of creatinine and urea in the blood.
ATN may be caused by sepsis leading to hypotension and renal hypoperfusion; ischemia due to renal hypoperfusion, like in hypovolemic shock; but it can also be due to exposure to toxins and nephrotoxic drugs which cause damage to the tubular cells. Such drugs include nephrotoxic antibiotics, contrast agents used during imaging studies, amphotericin B, and toxic heavy metals.
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