Angina pectoris
00:00 / 00:00
Videos
Notes
Angina pectoris
Cardiovascular system
Cardiovascular system
Cardiovascular system
Cardiovascular system
Cardiovascular system
Cardiovascular system
Assessments
Angina pectoris
Flashcards
0 / 6 complete
USMLE® Step 1 questions
0 / 5 complete
High Yield Notes
2 pages
Flashcards
Angina pectoris
of complete
Questions
USMLE® Step 1 style questions USMLE
of complete
*(-) decrease, (0) no effect, (+) increase
Memory Anchors and Partner Content
External References
First Aid
2022
2021
2020
2019
2018
Acebutolol p. 246
angina and p. 326
Angina
aortic stenosis p. 298
atherosclerosis p. 308
cilostazol/dipyridamole for p. 445
cocaine causing p. 594
contraindicated drugs p. 326, 329
drug therapy for p. 325, 326, 363
glycoprotein IIb/IIa inhibitors for p. 445
ischemic disease and p. 310
presentation p. 716
unstable/NSTEMI treatment p. 316
Intestinal angina p. 395
Angina pectoris
β -blockers for p. 247
Atherosclerosis p. 308
stable angina with p. 310
β -blockers p. 247
angina p. 326
Calcium channel blockers p. 325
angina p. 325
“Intestinal angina p. 395
myocardial O2 consumption/demand p. 292
angina treatment p. 326
Nitroglycerin p. 325
angina p. 310
Stable angina p. 310
Triptans p. 567
angina and p. 310
Variant angina p. 310
External Links
Transcript
Content Reviewers
Contributors
Angina comes from the latin angere, which means to strangle, and pectoris comes from pectus, meaning chest—so angina pectoris loosely translates to “strangling of the chest”, which actually makes a lot of sense, because angina pectoris is caused by reduced blood flow which causes ischemia to the heart muscle, or lack of oxygen to the heart, almost like the heart’s being strangled which causes terrible chest pain.
Stable angina or chronic angina is the most common type of angina and it usually happens when the patient has greater than or equal to 70% stenosis, meaning 70% of the artery is blocked by plaque buildup.
This small opening that blood flows through might be enough to supply the heart during rest, but if the body demands more blood and oxygen, like during exercise or stressful situations, the heart has to work harder, and therefore needs more blood and oxygen itself.
It’s during these time of exertion or emotional stress that people with stable angina have chest pain, since the blood flow isn’t meeting the metabolic demands of the heart muscle, or myocardium.
But the pain usually goes away with rest.
In the majority of cases, the underlying cause of stable angina is atherosclerosis of one or more the coronary arteries—arteries supplying blood to the heart muscles.
Other heart conditions that might lead to stable angina are ones that cause a thickened heart muscle wall, which would require more oxygen.
This increase in muscle size can be due to hypertrophic cardiomyopathy from a genetic cause, or as a result from the heart having to pump against higher pressures, as is the case in aortic stenosis, which is a narrowing of the aortic valve, or hypertension.
Sources
- "Robbins Basic Pathology" Elsevier (2017)
- "Harrison's Principles of Internal Medicine, Twentieth Edition (Vol.1 & Vol.2)" McGraw-Hill Education / Medical (2018)
- "Pathophysiology of Disease: An Introduction to Clinical Medicine 8E" McGraw-Hill Education / Medical (2018)
- "Pathogenesis of angina pectoris" undefined (1982)
- "Unstable angina pectoris: Pathogenesis and management" Current Problems in Cardiology (1989)
- "Management of Chronic Stable Angina" Critical Care Nursing Clinics of North America (2017)
For Institutions
Copyright © 2023 Elsevier, its licensors, and contributors. All rights are reserved, including those for text and data mining, AI training, and similar technologies.
Cookies are used by this site.
USMLE® is a joint program of the Federation of State Medical Boards (FSMB) and the National Board of Medical Examiners (NBME). COMLEX-USA® is a registered trademark of The National Board of Osteopathic Medical Examiners, Inc. NCLEX-RN® is a registered trademark of the National Council of State Boards of Nursing, Inc. Test names and other trademarks are the property of the respective trademark holders. None of the trademark holders are endorsed by nor affiliated with Osmosis or this website.
