Specialties
Library
Resources
For institutions
Free trial
Free trialSpecialties
Medicine (MD)Medicine (DO)Physician Assistant (PA)Nurse Practitioner (NP)Registered Nursing (RN)DentistryPharmacyOther Healthcare Professional
LibraryResources
EventsBlogGreeting CardsTestimonialsOsmosis Around the World

Questions or feedback?

Cardiovascular changes during hemorrhage
2,304 views
Summary of Cardiovascular changes during hemorrhage
When blood loss is high, arterial pressure rapidly decreases, followed by a series of compensatory cardiovascular responses to try to restore arterial pressure to normal and sustain perfusion to critical organs.

Videos

Notes

Physiology

Cardiovascular system

Anatomy and physiology
Cardiovascular system anatomy and physiology
Lymphatic system anatomy and physiology
Coronary circulation
Hemodynamics
Blood pressure, blood flow, and resistance
Pressures in the cardiovascular system
Laminar flow and Reynolds number
Resistance to blood flow
Compliance of blood vessels
Control of blood flow circulation
Microcirculation and Starling forces
Cardiac output
Measuring cardiac output (Fick principle)
Stroke volume, ejection fraction, and cardiac output
Cardiac contractility
Frank-Starling relationship
Cardiac preload
Cardiac afterload
Law of Laplace
Cardiac and vascular function curves
Altering cardiac and vascular function curves
Cardiac cycle
Cardiac cycle
Cardiac work
Pressure-volume loops
Changes in pressure-volume loops
Physiological changes during exercise
Cardiovascular changes during hemorrhage
Cardiovascular changes during postural change
Auscultation of the heart
Normal heart sounds
Abnormal heart sounds
Myocyte electrophysiology
Action potentials in myocytes
Action potentials in pacemaker cells
Excitability and refractory periods
Cardiac excitation-contraction coupling
Electrocardiography
Electrical conduction in the heart
Cardiac conduction velocity
ECG basics
ECG rate and rhythm
ECG intervals
ECG QRS transition
ECG axis
ECG normal sinus rhythm
ECG cardiac infarction and ischemia
ECG cardiac hypertrophy and enlargement
Blood pressure regulation
Baroreceptors
Chemoreceptors
Renin-angiotensin-aldosterone system

Assessments
Cardiovascular changes during hemorrhage

Flashcards

0 / 21 complete
High Yield Notes
6 pages
Chapter 20 Cardiovascular Physiology: Normal Variations of the Cardiovascular System Figure 20.1 Flowchart showing cardiovascular response to exercise. CARDIOVASCULAR CHANGES DURING HEMORRHAGE osms.it/cardiovascular-changes-hemorrhage ▪ Blood loss → ↓ arterial pressure → compensatory responses to restore arterial pressure ▫ Response mediated by baroreceptor reflex, renin-angiotensin-aldosterone system (RAAS), vascular actions Decrease in arterial pressure ▪ Hemorrhage → ↓ total blood volume → ↓ venous return to heart, ↓ right atrial pressure → ↓ cardiac output → ↓ Pa as a product of cardiac output, TPR Return of arterial pressure ▪ Baroreceptors in carotid sinus detect ↓ Pa → relay information to medulla via carotid sinus nerve → ↑ sympathetic outflow to heart, blood vessels; ↓ parasympathetic outflow to heart → ↑ heart rate, ↑ contractility, ↑ TPR, constriction of veins ▪ ↓ mean arterial pressure → ↓ perfusion to kidney → response via RAAS ▫ Kidney secretes renin from renal juxtaglomerular cells → ↑ angiotensin I production → converted to angiotensin II (causes arteriolar vasoconstriction, stimulates aldosterone secretion) ▪ Capillary changes favor fluid reabsorption ▫ ↑ sympathetic outflow to blood vessels, angiotensin II → arteriolar vasoconstriction → ↓ capillary hydrostatic pressure (Pc)→ restricts filtration out of capillaries, favors absorption OTHER RESPONSES IN HEMORRHAGE ▪ Hypoxemia (↓ arterial PO2): carotid, aortic bodies chemoreceptors sense ↓ PO2 → ↑ sympathetic outflow to blood vessels → ↑ vasoconstriction, TPR, Pa ▪ Cerebral ischemia: local ↑ PCO2 OSMOSIS.ORG 147
Cardiovascular changes during hemorrhage
Flashcards

Cardiovascular changes during hemorrhage

21 flashcards
Preview

Cerebral ischemia due to hemorrhage will cause a local (increase/decrease) in PCO2 and a decrease in pH, which activate chemoreceptors to increase sympathetic outflow and peripheral vasoconstriction in order to maintain cerebral perfusion.