Content Reviewers:Lisa Miklush, PhD, RNC, CNS, Jannah Amiel, MS, BSN, RN, Jodi Berndt, PhD, RN, CCRN-K, PCCN-K, CNE, CHSE, Gabrielle Proper, RN, BScN, MN
Dwayne Harris is a 52 year old African American male client presenting to the cardiac catheterization lab to undergo a percutaneous coronary intervention, also known as PCI.
Because his angina is no longer responding to treatment, his cardiologist recommended PCI for Mr. Harris.
This narrowing is caused by atherosclerosis, a lipid containing plaque that accumulates on artery walls.
Over time, the plaque build up reduces myocardial perfusion and causes ischemia as the demand for oxygen exceeds the supply.
Myocardial ischemia leads to a type of chest pain called angina, which can be either stable or unstable.
Stable angina usually occurs when atherosclerotic plaque is fixed to the artery wall and occludes at least 75 percent of the coronary artery, whereas with unstable angina, the plaque ruptures and almost completely occludes the artery lumen.
The clinical presentation helps to differentiate stable and unstable angina using the acronym OPQRST.
Q stands for the quality of pain, which often involves pressure, crushing, squeezing, or tightness. Pain is more severe with unstable angina.
R stands for radiation of pain, because it often radiates to the shoulders, arms, jaw, neck, or back.
Now, there are several non-modifiable and modifiable risk factors contributing to CAD and its complications.
Advanced age is the greatest risk factor, with men over 45 and women over 55 years of age being at most risk.
Other non-modifiable risk factors include biological male sex, family history of hypercholesterolemia, and belonging to African American, Native American, Native Hawaiian, and South Asian demographic groups.
Psychosocial factors, such as stress and depression can be risk factors for CAD too, due to their association with factors like smoking, physical inactivity, and obesity as well as their association with elevated systemic inflammation which contributes to atherosclerosis.
OK, treating CAD centers around lifestyle modifications, medication management and, if needed, coronary revascularization procedures.
Lifestyle modifications involve controlling modifiable risk factors.
Several medications are used to treat CAD as well.
Cholesterol lowering medications are prescribed to reduce atheroscleoritc plaque formation, including statins like atorvastatin, fibric acid derivatives such as fenofibrate, omega 3 fatty acids, bile acid sequestrants like colestipol, cholesterol absorption inhibitors such as ezetimibe, and PCSK9 inhibitors like evolocumab.
If stable angina isn’t effectively treated by lifestyle modifications and medication therapy, coronary revascularization procedures may be necessary.
If needed, a stent can be placed during PCI to keep the artery patent.
Alternatively, atherectomy devices are used to remove the plaque.
Coronary bypass grafting, or CABG, can be done instead of PCI.
OK, let’s get back to Mr. Harris and begin his assessment.
You introduce yourself, perform hand hygiene, and confirm his identity.
After asking if he feels ready for his procedure, he says he is anxious but hopeful it will help him have less chest pain when taking his daily walks.
Your assessment reveals warm, dry, intact skin with good turgor.
His oral temperature is 97.8°F, or 36.6°C.
Bowel sounds are active, and Mr. Harris confirms he hasn’t had anything to eat or drink since midnight.