Porphyria cutanea tarda
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Porphyria cutanea tarda
EXam 1
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Blood components
Erythropoietin
Blood groups and transfusions
Platelet plug formation (primary hemostasis)
Coagulation (secondary hemostasis)
Role of Vitamin K in coagulation
Clot retraction and fibrinolysis
Introduction to the immune system
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VDJ rearrangement
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Skin anatomy and physiology
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Iron deficiency anemia
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Porphyria cutanea tarda
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Vitamin K deficiency
Bernard-Soulier syndrome
Glanzmann's thrombasthenia
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Microcytic anemia: Pathology review
Non-hemolytic normocytic anemia: Pathology review
Intrinsic hemolytic normocytic anemia: Pathology review
Extrinsic hemolytic normocytic anemia: Pathology review
Macrocytic anemia: Pathology review
Heme synthesis disorders: Pathology review
Coagulation disorders: Pathology review
Platelet disorders: Pathology review
Mixed platelet and coagulation disorders: Pathology review
Thrombosis syndromes (hypercoagulability): Pathology review
Lymphomas: Pathology review
Leukemias: Pathology review
Plasma cell disorders: Pathology review
Myeloproliferative disorders: Pathology review
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IgG subclass deficiency
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Ruptured spleen
Immunodeficiencies: T-cell and B-cell disorders: Pathology review
Immunodeficiencies: Combined T-cell and B-cell disorders: Pathology review
Immunodeficiencies: Phagocyte and complement dysfunction: Pathology review
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Porphyria cutanea tarda
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A defect in which of the following steps of the heme biosynthesis pathway is most likely responsible for this patient’s symptoms?
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Porphyria cutanea tarda p. 432
Sporadic porphyria cutanea tarda p. 171
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Summary
Porphyria cutanea tarda is a rare metabolic disorder that is caused by an abnormality in the enzyme uroporphyrinogen decarboxylase. Symptoms of this disorder usually involve the skin, as it can lead to blisters and skin discoloration. Treatment for this disorder usually involves medications such as hydroxychloroquine and phlebotomy, as well as lifestyle changes such as avoiding alcohol.
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