Syncope: Clinical (To be retired)

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Syncope: Clinical (To be retired)

Medicine and surgery

Allergy and immunology

Antihistamines for allergies

Glucocorticoids

Cardiology, cardiac surgery and vascular surgery

Coronary artery disease: Clinical (To be retired)

Heart failure: Clinical (To be retired)

Syncope: Clinical (To be retired)

Hypertension: Clinical (To be retired)

Hypercholesterolemia: Clinical (To be retired)

Peripheral vascular disease: Clinical (To be retired)

Leg ulcers: Clinical (To be retired)

Adrenergic antagonists: Alpha blockers

Adrenergic antagonists: Beta blockers

ACE inhibitors, ARBs and direct renin inhibitors

Thiazide and thiazide-like diuretics

Calcium channel blockers

Lipid-lowering medications: Statins

Lipid-lowering medications: Fibrates

Miscellaneous lipid-lowering medications

Antiplatelet medications

Dermatology and plastic surgery

Hypersensitivity skin reactions: Clinical (To be retired)

Eczematous rashes: Clinical (To be retired)

Papulosquamous skin disorders: Clinical (To be retired)

Alopecia: Clinical (To be retired)

Hypopigmentation skin disorders: Clinical (To be retired)

Benign hyperpigmented skin lesions: Clinical (To be retired)

Skin cancer: Clinical (To be retired)

Endocrinology and ENT (Otolaryngology)

Diabetes mellitus: Clinical (To be retired)

Hyperthyroidism: Clinical (To be retired)

Hypothyroidism and thyroiditis: Clinical (To be retired)

Dizziness and vertigo: Clinical (To be retired)

Hyperthyroidism medications

Hypothyroidism medications

Insulins

Hypoglycemics: Insulin secretagogues

Miscellaneous hypoglycemics

Gastroenterology and general surgery

Gastroesophageal reflux disease (GERD): Clinical (To be retired)

Peptic ulcers and stomach cancer: Clinical (To be retired)

Diarrhea: Clinical (To be retired)

Malabsorption: Clinical (To be retired)

Colorectal cancer: Clinical (To be retired)

Diverticular disease: Clinical (To be retired)

Anal conditions: Clinical (To be retired)

Cirrhosis: Clinical (To be retired)

Breast cancer: Clinical (To be retired)

Laxatives and cathartics

Antidiarrheals

Acid reducing medications

Hematology and oncology

Anemia: Clinical (To be retired)

Anticoagulants: Warfarin

Anticoagulants: Direct factor inhibitors

Antiplatelet medications

Infectious diseases

Pneumonia: Clinical (To be retired)

Urinary tract infections: Clinical (To be retired)

Skin and soft tissue infections: Clinical (To be retired)

Protein synthesis inhibitors: Aminoglycosides

Antimetabolites: Sulfonamides and trimethoprim

Miscellaneous cell wall synthesis inhibitors

Protein synthesis inhibitors: Tetracyclines

Cell wall synthesis inhibitors: Penicillins

Miscellaneous protein synthesis inhibitors

Cell wall synthesis inhibitors: Cephalosporins

DNA synthesis inhibitors: Metronidazole

DNA synthesis inhibitors: Fluoroquinolones

Herpesvirus medications

Azoles

Echinocandins

Miscellaneous antifungal medications

Anti-mite and louse medications

Nephrology and urology

Chronic kidney disease: Clinical (To be retired)

Kidney stones: Clinical (To be retired)

Urinary incontinence: Pathology review

ACE inhibitors, ARBs and direct renin inhibitors

PDE5 inhibitors

Adrenergic antagonists: Alpha blockers

Neurology and neurosurgery

Stroke: Clinical (To be retired)

Lower back pain: Clinical (To be retired)

Headaches: Clinical (To be retired)

Migraine medications

Pulmonology and thoracic surgery

Asthma: Clinical (To be retired)

Chronic obstructive pulmonary disease (COPD): Clinical (To be retired)

Lung cancer: Clinical (To be retired)

Antihistamines for allergies

Bronchodilators: Beta 2-agonists and muscarinic antagonists

Bronchodilators: Leukotriene antagonists and methylxanthines

Pulmonary corticosteroids and mast cell inhibitors

Rheumatology and orthopedic surgery

Joint pain: Clinical (To be retired)

Rheumatoid arthritis: Clinical (To be retired)

Lower back pain: Clinical (To be retired)

Anatomy clinical correlates: Clavicle and shoulder

Anatomy clinical correlates: Arm, elbow and forearm

Anatomy clinical correlates: Wrist and hand

Anatomy clinical correlates: Median, ulnar and radial nerves

Anatomy clinical correlates: Bones, joints and muscles of the back

Anatomy clinical correlates: Hip, gluteal region and thigh

Anatomy clinical correlates: Knee

Anatomy clinical correlates: Leg and ankle

Anatomy clinical correlates: Foot

Acetaminophen (Paracetamol)

Non-steroidal anti-inflammatory drugs

Glucocorticoids

Opioid agonists, mixed agonist-antagonists and partial agonists

Antigout medications

Osteoporosis medications

Assessments

Syncope: Clinical (To be retired)

USMLE® Step 2 questions

0 / 3 complete

Questions

USMLE® Step 2 style questions USMLE

of complete

A 76-year-old man comes to the office because of a brief loss of consciousness 2 hours ago. He was having dinner with two friends when he looked "pale" and slumped in his chair. He regained consciousness after about two minutes. He has a history of  hypertension and coronary artery disease, which has been treated with two stents. His temperature is 36.8°C (98°F), pulse is 145/min, respirations are 18/min, and blood pressure is 90/68 mm Hg. Cardiac auscultation shows no murmurs. An ECG is obtained and the rhythm strip is shown below. Which of the following is the most likely diagnosis?


Transcript

Content Reviewers

Rishi Desai, MD, MPH

Contributors

Antonella Melani, MD

Gil McIntire

Tanner Marshall, MS

Justin Ling, MD, MS

Syncope, or fainting, is when a person loses consciousness and muscle strength. It usually comes on quickly, doesn’t last long, and there’s usually a spontaneous recovery requiring no resuscitation.

It’s caused by a decrease in blood flow to the brain, usually due to low blood pressure.

There’s also presyncope, which is near loss of consciousness with lightheadedness, muscular weakness, blurred vision, and feeling faint without actually fainting.

Presyncope can lead to syncope, so you can think of it as a spectrum of the disease.

Recognizing symptoms of presyncope may allow to act fast and prevent evolution of the episode into a full faint.

The immediate treatment of an individuals with syncope or presyncope starts with laying the individual supine, with legs elevated if possible to help venous return to the heart and restore adequate brain perfusion.

Then, you should assess vital signs, namely a pulse and evidence of respiration, to distinguish cardiac arrest from syncope, and call for additional help if needed.

Finally, you should attempt to arouse the individual without trying to raise them up until they’re ready.

Ok so once the individual has regained consciousness, the next step is to identify the cause.

Neurocardiogenic, vasovagal, and reflex syncope are the most common causes of syncope, and this is a benign condition triggered by parasympathetic activation resulting in vagus nerve discharge.

This discharge may in turn be triggered by urination, defecation, coughing, prolonged standing, or a stressful event like seeing blood and needles.

Summary

Elsevier

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