Essential fructosuria
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Essential fructosuria
Obesity
Obesity
Childhood nutrition and obesity: Information for patients and families (The Primary School)
Glycolysis
Citric acid cycle
Physiological changes during exercise
Essential fructosuria
Hereditary fructose intolerance
Galactosemia
Pyruvate dehydrogenase deficiency
Lactose intolerance
Glycogen storage disease type III
Glycogen storage disease type IV
Glycogen storage disease type V
Krabbe disease
Hartnup disease
Alkaptonuria
Ornithine transcarbamylase deficiency
Abetalipoproteinemia
Familial hypercholesterolemia
Hyperlipidemia
Carbohydrates and sugars
Fats and lipids
Proteins
Vitamin K deficiency
Vitamin D deficiency
Excess Vitamin A
Excess Vitamin D
Folate (Vitamin B9) deficiency
Niacin (Vitamin B3) deficiency
Vitamin B12 deficiency
Vitamin C deficiency
Beriberi
Iodine deficiency
Zinc deficiency
Marasmus
Kwashiorkor
Fat-soluble vitamin deficiency and toxicity: Pathology review
Zinc deficiency and protein-energy malnutrition: Pathology review
Water-soluble vitamin deficiency and toxicity: B1-B7: Pathology review
Eating disorders: Clinical
Placebo effect and masking
Prader-Willi syndrome
Hunger and satiety
Development of the digestive system and body cavities
Key Takeaways
Essential fructosuria is a benign autosomal recessive disorder in which there is a deficiency caused by a deficiency fructokinase enzyme. People with essential fructosuria don't metabolize fructose properly after ingesting a fructose-rich diet, causing it to be in urine, hence the name fructosuria.