Acute tubular necrosis
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Acute tubular necrosis
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Renal system
Renal system
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Acute tubular necrosis
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Acute tubular necrosis
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| Laboratory value | Result | ||
| Urinalysis | |||
| On admission | Day 2 | Day 8 | |
| Creatinine | 1.1 mg/dL | 3.5 mg/dL | 2.3 mg/dL |
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First Aid
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Acute tubular necrosis p. 626
Aminoglycosides p. 188
acute tubular necrosis p. 627
Cisplatin p. 449
acute tubular necrosis p. 627
Granular casts p. 618
acute tubular necrosis p. 627
Heart failure p. 318
acute tubular necrosis with p. 626
Hemoglobinuria
acute tubular necrosis and p. 627
Hemorrhage
acute tubular necrosis p. 627
Hypotension
acute tubular necrosis with p. 627
Ischemia p. 208, 691
acute tubular necrosis from p. 626
Lead poisoning p. 427, 432
acute tubular necrosis p. 626
Myoglobinuria
acute tubular necrosis p. 627
Sepsis
acute tubular necrosis p. 627
Shock p. 319
acute tubular necrosis p. 627
Tubular necrosis p. 618, 626, 626
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Summary
Acute tubular necrosis (ATN) is a type of acute kidney injury (AKI) that results in the sudden and rapid death of tubular cells in the kidneys. This can lead to a decrease in urine production and an increase in the levels of creatinine and urea in the blood.
ATN may be caused by sepsis leading to hypotension and renal hypoperfusion; ischemia due to renal hypoperfusion, like in hypovolemic shock; but it can also be due to exposure to toxins and nephrotoxic drugs which cause damage to the tubular cells. Such drugs include nephrotoxic antibiotics, contrast agents used during imaging studies, amphotericin B, and toxic heavy metals.
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