Atopic dermatitis: Nursing process (ADPIE)

Atopic dermatitis: Nursing process (ADPIE)

Nursing

Nursing

Aortic aneurysm: Nursing process (ADPIE)
Coronary artery disease (CAD) and angina pectoris: Nursing process (ADPIE)
Hypertension: Nursing process (ADPIE)
Left-sided heart failure: Nursing process (ADPIE)
Myocardial infarction (MI): Nursing process (ADPIE)
Pericardial effusion and cardiac tamponade: Nursing process (ADPIE)
Rheumatic heart disease: Nursing process (ADPIE)
Diabetes insipidus: Nursing process (ADPIE)
Diabetes mellitus (DM): Nursing process (ADPIE)
Diabetic ketoacidosis (DKA): Nursing process (ADPIE)
Hyperthyroidism: Nursing process (ADPIE)
Hyperosmolar hyperglycemic state (HHS): Nursing process (ADPIE)
Hypothyroidism: Nursing process (ADPIE)
Syndrome of inappropriate antidiuretic hormone (SIADH): Nursing process (ADPIE)
Epistaxis: Nursing process (ADPIE)
Eye injury: Nursing process (ADPIE)
Glaucoma: Nursing process (ADPIE)
Tonsillitis: Nursing process (ADPIE)
Appendicitis: Nursing process (ADPIE)
Celiac disease: Nursing process (ADPIE)
Cirrhosis: Nursing process (ADPIE)
Gastroesophageal reflux disease (GERD): Nursing process (ADPIE)
Hiatal hernia: Nursing process (ADPIE)
Pancreatitis: Nursing process (ADPIE)
Peptic ulcer disease (PUD): Nursing process (ADPIE)
Acute kidney injury (AKI): Nursing process (ADPIE)
Benign prostatic hyperplasia (BPH): Nursing process (ADPIE)
Urinary tract infections (UTIs): Nursing process (ADPIE)
Urinary incontinence - Stress: Nursing process (ADPIE)
Age-related physiological changes: Nursing process (ADPIE)
Hemophilia: Nursing process (ADPIE)
Leukemia: Nursing process (ADPIE)
Sickle cell disease: Nursing process (ADPIE)
Anaphylaxis: Nursing process (ADPIE)
Lyme disease: Nursing process (ADPIE)
Atopic dermatitis: Nursing process (ADPIE)
Frostbite: Nursing process (ADPIE)
Methicillin-resistant Staphylococcus aureus (MRSA): Nursing process (ADPIE)
Pressure injury: Nursing process (ADPIE)
Hyperbilirubinemia: Nursing process (ADPIE)
Placenta previa: Nursing process (ADPIE)
Placental abruption: Nursing process (ADPIE)
Prolapsed umbilical cord: Nursing process (ADPIE)
Anxiety disorders: Nursing process (ADPIE)
Eating disorders: Nursing process (ADPIE)
Acute compartment syndrome: Nursing process (ADPIE)
Fractures: Nursing process (ADPIE)
Gout: Nursing process (ADPIE)
Musculoskeletal injuries: Nursing process (ADPIE)
Rheumatoid arthritis (RA): Nursing process (ADPIE)
Meningitis: Nursing process (ADPIE)
Parkinson disease: Nursing process (ADPIE)
Stroke: Nursing process (ADPIE)
Trigeminal neuralgia: Nursing process (ADPIE)
Seizure disorder: Nursing process (ADPIE)
Breast cancer: Nursing process (ADPIE)
Tumor lysis syndrome (TLS): Nursing Process (ADPIE)
Bronchiolitis and respiratory syncytial virus (RSV): Nursing process (ADPIE)
Chickenpox (Varicella): Nursing process (ADPIE)
Conjunctivitis: Nursing process (ADPIE)
Hydrocephalus: Nursing process (ADPIE)
Poisoning: Nursing process (ADPIE)
Pyloric stenosis: Nursing process (ADPIE)
Reye syndrome: Nursing process (ADPIE)
Peripheral arterial disease (PAD): Nursing process (ADPIE)
Peripheral venous disease (PVD): Nursing process (ADPIE)
Candidiasis: Nursing process (ADPIE)
Gonorrhea and chlamydia: Nursing process (ADPIE)
Pelvic inflammatory disease (PID): Nursing process (ADPIE)
Asthma: Nursing process (ADPIE)
Bacterial pneumonia: Nursing process (ADPIE)
Carbon monoxide poisoning: Nursing process (ADPIE)
Chronic obstructive pulmonary disease (COPD): Nursing process (ADPIE)
Epiglottitis: Nursing process (ADPIE)
Foreign body aspiration and upper airway obstruction: Nursing process (ADPIE)
Laryngotracheobronchitis (LTB) and croup: Nursing process (ADPIE)
Smoke inhalation injury: Nursing process (ADPIE)
Venous thromboembolism (VTE): Nursing process (ADPIE)

Notes

ATOPIC DERMATITIS

KEY POINTS
NOTES
PATIENT REPORT
  • 2-year-old boy
  • Red rash to scalp, cheeks, chest
  • Cracks and oozing
  • Diagnosis: atopic dermatitis

PATHOPHYSIOLOGY
  • Atopic dermatitis
    • Chronic inflammatory skin disease 
  • Causes
    • Not fully understood
    • Skin barrier abnormalities
    • Dysfunction of immune system
  • Risk factors
    • Younger age
    • Atopy
    • Family history
    • Mutations in filaggrin gene
    • Contact with triggers
  • Signs and symptoms
    • Dry, scaly skin
    • Itchiness
    • Patches of red, swollen, and easily irritated skin
    • Erosions, oozing, crusting
  • Complications
    • Secondary bacterial, fungal, or viral  infections

DIAGNOSIS AND TREATMENT
  • Diagnosis
    • History
    • Physical assessment
    • Laboratory tests
  • Treatment
    • Moisturizers
    • Antihistamines
    • Topical medications
    • Avoid triggers

ASSESSMENT
  • Rash began 1 week ago
  • Dry, scaly, red skin patches on cheeks and scalp
  • Trunk is dry and irritated
  • Lesions weep and bleed where scratched 
  • Temperature: 98.9 F (37 C)
  • Heart rate: 80
  • Respiratory rate: 28
  • Lungs clear
  • Oxygen saturation: 99% room air

NURSING DIAGNOSES
  • Impaired skin integrity related to inflammation
  • Risk for infection elated to broken skin 
  • Impaired skin comfort related to pruritus
  • Readiness for enhanced parental knowledge

PLANNING
  • Decrease inflammation
  • Less pruritus
  • No evidence of infection

IMPLEMENTATION
  • Demonstrate how to clean rash
  • Show how to apply medications
  • Remind to watch for triggers
  • Stress when to seek emergency medical care

EVALUATION
  • Inflammation receding 
  • Less itching
  • No signs of infection
  • Enjoying bath time

Transcript

Watch video only

Kameron Jones is a 2-year-old male who presents to your pediatric clinic accompanied by his mother.

Mrs. Jones explains that Kameron has developed a red rash on his scalp, cheeks, and chest that has gotten worse over the past week.

Yesterday, she noticed that some of those areas of skin looked red and developed cracks that began to ooze.

She says Kameron has been scratching at his chest constantly, and he’s been more irritable, since the rash appeared.

The pediatrician diagnoses atopic dermatitis, and Kameron will begin treatment today.

Atopic dermatitis, also known as eczema, is a chronic inflammatory skin disease that occurs most frequently in children, ages five and under, but may also affect adults, especially those with a predisposition towards allergic diseases, including asthma, allergic rhinitis, and conjunctivitis.

Now, the exact cause of atopic dermatitis is not fully understood, however it seems to be a mix of skin barrier abnormalities and dysfunction in the immune system.

In regards to skin barrier abnormalities, atopic dermatitis is associated with a mutation in the filaggrin gene.

Filaggrin is a protein that binds to keratin and contributes to the formation of the skin barrier, so individuals with atopic dermatitis have a slightly porous skin barrier, which sets them up for allergens or microbes to get under the skin.

In regards to dysfunction in the immune system, atopic dermatitis is associated with atopy, which is a genetic predisposition to develop allergies due to an increased inflammatory response to triggers, such as allergens or irritant substances.

There are some risk factors for atopic dermatitis.

Unfortunately, some of them are non-modifiable, including younger age, atopy, family history, and mutations in the filaggrin gene.

On the other hand, modifiable risk factors include contact with triggers, including allergens like pollen, mold, or animal dander, as well as a certain kind of soap or detergent, or an irritant like tobacco smoke.

Clients with eczema typically present with dry, scaly skin that’s very itchy, as well as patches of red, swollen, and easily irritated skin, especially on the face and scalp, but sometimes also on the insides of the elbows or the back of the knees.

When scratched, these sensitive patches of skin may develop erosions, oozing, and crusting.

An important complication that may affect this disrupted skin barrier is secondary bacterial infections, especially staphylococcal and streptococcal infections, but also fungal infections, such as tinea, and viral infections, most often from herpes simplex virus.

Diagnosis of atopic dermatitis is based on the client’s history and physical exam.

Laboratory tests may show increased eosinophils and serum IgE levels, while allergy testing can help identify the specific allergens that should be avoided.

There is no cure for atopic dermatitis, so treatment aims at reducing the symptoms by keeping the skin hydrated with moisturizers, minimizing itching with oral antihistamines, and dampening the inflammatory reaction with topical medications, such as steroid creams like hydrocortisone or calcineurin inhibitors like tacrolimus.

Finally, clients should try to avoid exposure to triggers. Let’s get back to assess Kameron and see how he’s doing.

You wash your hands, introduce yourself to him and his mother, and confirm his identity.

Kameron is sitting in his mother’s lap and appears uncomfortable as he scratches his chest through his shirt.

You ask Mrs. Jones when she first noticed the rash.

She says it appeared on his chest about a week ago, and that she thought something had scratched him, but now that it’s also affecting his face and scalp.

She’s concerned it is getting worse and wonders if it is contagious.

Sources

  1. "Ackley and Ladwig’s Nursing Diagnosis Handbook: An Evidence-Based Guide to Planning Care, 13th edition" Mosby (2022)
  2. "Pathophysiology of atopic dermatitis: Clinical implications" Allergy Asthma Proc (2019)
  3. "Harrison’s Principles of Internal Medicine, 21st edition" McGraw Hill / Medical (2022)
  4. "Daily Moisturization for Atopic Dermatitis: Importance, Recommendations, and Moisturizer Choices" The Journal for Nurse Practitioners (2021)
  5. "Molecular Mechanisms of Atopic Dermatitis Pathogenesis" Int J Mol Sci (2021)
  6. "Critical Care Nursing: Diagnosis and Management, 9th edition" Elsevier (2021)
  7. "Health Assessment for Nursing Practice, 7th edition" Elsevier (2021)