Calcaneal spurs develop as the body’s response to inflammation of a tendon or ligament at its attachment point to the bone. In plantar heel spurs, repetitive microtrauma or traction of the plantar fascia can result in an inflammatory reaction that triggers the formation of new bone. In many cases, heel spurs coexist with inflammation of the plantar fascia, or plantar fasciitis, but it is unclear whether spurs cause plantar fasciitis or represent a secondary response to inflammation.
Risk factors for heel spurs include conditions that put more pressure on the heel, such as
obesity, prolonged standing, working on hard surfaces, overtraining, and
flat feet. As individuals
age, the heel’s protective fat pad becomes thinner and the plantar fascia becomes less flexible, increasing the risk of both heel spurs and plantar fasciitis. Some individuals may have a
genetic predisposition to generate heel spurs from levels of
stress insufficient to initiate
bone growth in others. Finally, individuals with bone and joint disorders such as
osteoarthritis,
rheumatoid arthritis,
gout, and
ankylosing spondylitis are also more likely to develop heel spurs.