Lyme disease: Nursing process (ADPIE)

Lyme disease: Nursing process (ADPIE)

test 4

test 4

Diabetes insipidus
Diabetes mellitus: Clinical
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Diabetes mellitus: Pathology review
Graves disease
Hyperthyroidism: Nursing process (ADPIE)
Hyperthyroidism
Hyperthyroidism: Pathology review
Hyperparathyroidism
Hyperparathyroidism: Nursing
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Sickle cell disease: Nursing process (ADPIE)
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Food allergies and EpiPens: Information for patients and families (The Primary School)
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Appendicitis: Pathology review
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Human papillomavirus
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The flu vaccine: Information for patients and families
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Legionella pneumophila (Legionnaires disease and Pontiac fever)
Borrelia burgdorferi (Lyme disease)
Lyme disease: Nursing process (ADPIE)
Lyme Disease
Meningitis: Nursing process (ADPIE)
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Varicella zoster virus

Notes

LYME DISEASE

KEY POINTS
NOTES
PATIENT REPORT
  • 32-year-old man
  • Presents to PCP with bull's eye rash
  • Recent camping
  • Reports 
    • Fatigue
    • Fever
    • Chills
    • Muscle aches
    • Joint pain & stiffness

PATHOPHYSIOLOGY
  • Cause
    • Borrelia bacteria
      • Most commonly Borrelia burgdorferi 
      • Zoonotic disease requiring a vector for transmission 
      • Transmitted by Ixodes ticks (deer ticks) 
        • Common in wooded areas, brush, marshes, and tall grass 
      • Ticks acquire the bacteria from infected animals 
        • Transmit bacteria to humans via saliva 
  • Risk factors
    • Outdoor activities in grassy or wooded areas  
    • Occupations
      • Landscaping
      • Farming
      • Railroad work 
    • Wearing skin-exposing clothing 
    • More common in spring and summer due to tick activity
  • Stages
    • Stage 1
      • Early localized (within 1 month) 
        • Flu-like symptoms 
        • Erythema migrans
          • Painless, expanding red rash
          • Bull’s-eye appearance
    • Stage 2
      • Early disseminated (weeks to months) 
        • Transient migratory arthritis 
        • Carditis
          • Risk of arrhythmias  
        • Meningoencephalitis
        • Facial nerve palsy
    • Stage 3
      • Late disseminated (months to years) 
        • Chronic arthritis 
        • Chronic encephalopathy
        • Gait abnormalities
        • Fatigue
        • Memory loss
        • Personality changes 
        • Peripheral nerve damage

DIAGNOSIS AND TREATMENT
  • Diagnosis
    • History
    • Physical assessment
    • Serologic testing 
      • Enzyme-linked immunosorbent assay (ELISA)
      • Confirmatory test
        • Western blot 
        • Diagnosis confirmed only if both tests are positive 
      • Inflammatory markers 
    • ECG
    • Lumbar puncture 
  • Treatment
    • Antibiotics 
    • Supportive care 
    • Temporary pacemaker for arrhythmias 

ASSESSMENT
  • Patient recently returned from a camping trip 
  • Found and removed a tick from his thigh 
  • Noticed a large, painless rash resembling a bull’s-eye 
    • Rash has expanded  
    • Raised edges and flat center 
  • Vital signs 
    • Temperature: 101°F (38.3°C) temporal 
    • Heart Rate: 100 bpm 
    • Respiratory Rate: 18 breaths/min 
    • Blood Pressure: 122/78 mmHg 
    • SpO2: 99% on room air 
    • Pain rated 5/10 
      • Located in joints

NURSING DIAGNOSES
  • Elevated body temperature related to infectious process
  • Fatigue related to illness
  • Pain related to joint inflammation
  • Readiness for enhanced health management related to new therapeutic treatment regimen

PLANNING
  • By follow-up appointment 
    • Patient's temperature will be within normal limits
    • Patient will report less fatigue and joint pain and stiffness
    • Patient will adhere to treatment regimen
      • Prevent Lyme disease in future

IMPLEMENTATION
  • Prescribed
    • Doxycycline 
    • NSAID  
  • Advised to contact HCP 
    • Neck stiffness 
    • Increased joint pain 
    • Lightheadedness 
    • Palpitations 
  • Prevention education 
    • Cover skin during outdoor activities 
    • Use insect repellent containing DEET 
    • Perform full-body tick checks after exposure 

EVALUATION
  • Completed full course of doxycycline 
  • Reports significant improvement in symptoms 
  • Vital signs 
    • Temperature: 98.9°F (37.1°C) temporal 
    • Pain rated 2/10 
  • Patient more conscientious about tick-bite prevention 

Transcript

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Jacob Myers is a 32-year-old male who presents to his primary care provider after discovering a painless bulls-eye looking rash on his thigh that has gotten bigger over the past three days.

He also reports symptoms of fatigue, fever, chills, muscle aches along with joint pain and stiffness that started three days after he returned from a week-long camping trip.

The provider suspects that Jacob has Lyme disease. Lyme disease is an infectious disease that’s caused by a bacterial species called Borrelia, most often Borrelia burgdorferi.

The Borrelia species are all spirochetes, which means that they are long, thin, and spiral-shaped.

Now, Lyme disease is a zoonotic disease, meaning that it can infect a wide range of animals, including deer, cattle, and rodents, but it isn’t known to spread directly from animals to humans, but instead it needs a vector, meaning some sort of intermediate organism to spread from the animal to the human.

In Lyme disease, the vector is the Ixodes tick, which is often found in wooded areas, thick brush, marshes, and tall grass.

Now, the transmission of Lyme disease starts when the Ixodes tick feeds on the blood from an infected animal host.

If this tick then feeds on the blood from a human, it will transmit the bacteria with their saliva.

Once the bacteria is in a human's bloodstream, it can disseminate to distant tissues, particularly the skin, joints, and heart, causing inflammation and damage.

Now, there are some factors that may put a client at risk of Lyme disease, such as outdoor activities in wooded or grassy areas, like hiking and hunting, as well as some occupations like landscaping, farming, and railroad work.

The risk is also increased when clients wear clothing that exposes the skin. In addition, Lyme disease is more common during spring and summer, as ticks typically prefer warm temperatures.

Now, symptoms of Lyme disease can progress through three stages. The first is called the early localized stage, which starts within 1 month of the tick bite.

Clients in this stage might report flu-like symptoms like fever, headache, fatigue and lymphadenopathy, as well as muscle or joint pain.

A characteristic symptom of the early localized stage of Lyme disease is erythema migrans, which is a painless, red skin lesion that slowly expands over a few days around the site of the tick bite, developing a bulls-eye shape.

Now, if the disease is not treated at its first stage, the bacteria can disseminate to places like the joints, heart, and brain.

The second stage of Lyme disease is the early disseminated stage which usually occurs within weeks to months from the initial infection, and can cause complications like arthritis, most often involving the knees, wrists, and ankles.

This is called transient migratory arthritis, which is when arthritis resolves in one joint and moves to affect another.

Another complication at this stage is carditis, or inflammation of the heart, which increases the risk for arrhythmias, such as atrioventricular block, where electrical impulses can’t pass from the atria to the ventricles.

Finally, the early disseminated stage can also cause neurological complications like meningoencephalitis and facial nerve palsy, where there’s a weakness on one side of the face.

Ultimately, clients can reach the third stage of Lyme disease, also called the late disseminated stage, which usually develops months or sometimes even years after the initial infection.

The third stage typically presents with chronic arthritis, as well as neurological manifestations like chronic encephalopathy, gait abnormalities, fatigue, memory loss, and personality changes.

Finally, the third stage of Lyme disease can cause damage to peripheral nerves, which manifests as pain, numbness, and muscle weakness mainly involving the limbs.

Now, diagnosis of Lyme disease is typically based on history and physical examination.

Diagnosis can be confirmed with blood tests, which look for antibodies against Borrelia.

This starts with enzyme-linked immunosorbent assay, or ELISA, and is then confirmed with Western blot.