Valvular heart disease: Nursing
Introduction0:00–0:25
Valvular heart disease refers to damage or defect involving one or more heart valves, and it can be divided into stenosis or narrowing of the valvular orifice; as well as regurgitation or insufficiency, and prolapse, in which the valvular leaflets fail to close adequately.
Now, let’s go over some anatomy and physiology. If we take a look at a cross-section of the heart.
Physiology0:25–2:02
Here, we can see the outer layer called the epicardium; the middle layer called the myocardium, and finally, a very thin inner layer called the endocardium.
Now, the endocardium folds over the heart openings, forming the valvular leaflets that separate the heart chambers, so the atria and ventricles.
There are two atrioventricular valves; the mitral valve separates the left atrium from the left ventricle, while the tricuspid valve separates the right atrium from the right ventricle.
These valves are connected to the papillary muscles, and chordae tendinae of the heart. So when the heart is relaxed during diastole, they are open, enabling the ventricles to fill up with blood.
On the flip side, when the heart contracts during systole, the valves close, thereby preventing the blood flow from the ventricles back into the atria.
Then, there are two semilunar valves; the aortic valve separates the left ventricle from the aorta, while the pulmonary valve separates the right ventricle from the pulmonary artery.
Now, in contrast to the atrioventricular valves, the semilunar valves close after ventricular contraction during systole, when the heart starts to relax in diastole, thereby preventing the backflow of the blood from the arteries into the ventricles.
Alright, so valvular heart disease is caused by damage or defects involving one or more heart valves. Rheumatic fever, which is a delayed inflammatory condition that typically occurs two to four weeks after a strep throat infection from Streptococcus pyogenes, also referred to as group A beta hemolytic streptococcus or GAS for short.
Causes & risk factors2:02–3:23
Other important causes of valvular heart disease include infective endocarditis, atherosclerosis, myocardial infarction, and dilated cardiomyopathy.
Finally, valvular heart disease can have congenital causes, such as congenital valve disease characterized by malformation defects during fetal life, as well as Marfan syndrome, which is an inherited connective tissue disorder that can affect a client’s skeleton, blood vessels, eyes, and lungs.
Now, risk factors for valvular heart disease include genetic predisposition and family history; as well as older age; and a history of strep throat or rheumatic fever, infectious endocarditis, cardiomyopathy, and valvular disease.
Other important risk factors include congenital heart diseases; autoimmune conditions, such as rheumatoid arthritis and systemic lupus erythematosus; and radiation exposure.
So the pathology depends on the type of valvular heart disease, as well as the affected valve. Let’s start with mitral stenosis.
Pathology3:23–5:54
In mitral stenosis, the leaflets might fuse together, impairing the opening of the mitral valve. The impaired opening will make it harder for blood to flow from the left atrium to the left ventricle.
As a result, the pressure in the left atrium increases; so to compensate, the left atrium undergoes hypertrophy, as well as dilation to accommodate the extra blood.
Over time, this compensation won’t be enough, resulting in backflow of blood in the pulmonary circulation. This can result in pulmonary edema, as well as pulmonary hypertension, which can ultimately make it harder for the right ventricle to pump blood into the pulmonary artery.
Over time, the right ventricle can undergo hypertrophy, and ultimately fail, which is also known as right-sided heart failure.
Next up is mitral valve regurgitation, in which the valves fail to close completely during systole. A type of mitral valve regurgitation is mitral valve prolapse, where the leaflets of the mitral valve bulge back into the left atrium during systole.
As a result, blood flows back from the left ventricle into the left atrium. Over time, this volume overload leads to atrial dilation, and the buildup of blood in the pulmonary circulation.
This results in pulmonary hypertension, which in turn increases the pressure in the right ventricle, causing it to undergo hypertrophy, and eventually leads to right-sided heart failure.
With aortic stenosis, the three leaflets of the aortic valve become stiff and often calcified. As a result, they can’t open fully, making it harder for blood to flow from the left ventricle into the aorta.
To compensate, the left ventricle undergoes hypertrophy and dilation to accommodate the extra blood. Over time, this compensation won’t be enough, resulting in left ventricular failure, which is followed by subsequent accumulation of blood in the left atrium and pulmonary circulation.
Finally, in aortic regurgitation, the valve doesn’t properly close during diastole, therefore blood leaks back from the aorta into the left ventricle.
To compensate, the left ventricle undergoes hypertrophy and dilation. Over time, when the left ventricle cannot compensate anymore, the client develops left ventricular heart failure.
Clinical manifestations5:54–7:14
Initially, valvular heart disease may not have clinical manifestations, so clients are generally asymptomatic for a long period, even for decades.
But, when symptoms do develop, they usually include dyspnea, orthopnea, fatigue, and angina. Upon auscultation, clients may present various heart murmurs depending on the affected valve and disease.
Mitral stenosis is characterized by a low-pitched mid to late diastolic murmur; while in mitral valve prolapse, there’s a mid-systolic click, sometimes followed by a systolic murmur.
On the other hand, in mitral regurgitation, there’s a pansystolic murmur, often referred to as a high-pitched holosystolic murmur, and an audible S3 heart sound.
Next up, aortic stenosis is characterized by a systolic crescendo-decrescendo murmur and decreased S2 sound; and lastly, aortic regurgitation is associated with a decrescendo diastolic murmur.
In severe cases, clients might develop complications like heart failure, leading to peripheral edema or syncope; while others may experience atrial fibrillation, and even cardiogenic shock.
Diagnosis7:14–7:42
The diagnosis of valvular heart disease starts with the client’s history and physical assessment, including electrocardiogram or ECG assessment, and exercise tolerance testing, or ETT for short.
Other important diagnostic methods include chest X-ray, as well as a Doppler, transthoracic or transesophageal echocardiography, as well as a stress echocardiography, or cardiac catheterization to evaluate the affected valve.
Treatment7:42–8:25
Now, the treatment of valvular heart disease can involve three approaches. First, pharmacological therapy includes anticoagulants, and severe cases may require medications used in the treatment of heart failure, such as ACE inhibitors and beta-blockers.
Second, minimally invasive procedures include percutaneous transluminal balloon valvuloplasty for valvular stenosis, in which the valve is dilated using a balloon; and percutaneous valve replacement, in which the affected valve is replaced with a new one.
And third, surgical therapy involves open-heart surgery with valve repair or replacement. Alright, let’s look at the nursing care you’ll be providing for a client with valvular stenosis.
Management of care8:25–10:40
Your priority nursing goals are to assist in maintaining your client’s cardiopulmonary function, and monitor for complications after surgery.
Okay, begin by assessing your client’s cardiopulmonary function by instituting continuous cardiac monitoring and pulse oximetry.
Then, check their vital signs; auscultate their heart and lung sounds; monitor their mental status; and keep track of their intake and output.
Immediately report to the health care provider if your client has an irregular heart rhythm, or signs of decreased cardiac output, including hypotension, tachypnea, shortness of breath, decreased SpO2, pulmonary crackles, decreased LOC, or decreased urinary output.
Administer the prescribed medications, such as diuretics to promote fluid excretion, antidysrhythmics for irregular heart rhythm.
Provide supplemental oxygen and assist your client into a high Fowler’s position to aid in breathing. Lastly, promote bed rest and institute fall precautions, and prepare your client for surgical intervention.
Now, let's go over providing care after a percutaneous transluminal balloon valvuloplasty. First assist your client into a comfortable position with the head of the bed flat or raised no more than 10 to 20 degrees.
Maintain bed rest; watch your client’s femoral access site for signs of bleeding; and perform frequent checks of vital signs, pulses distal to the insertion site, as well as neurovascular checks.
Be sure to apply the prescribed manual compression device and keep the affected limb straight. Assess their pain level and administer the prescribed analgesics, as needed.
Immediately report to the healthcare provider increased bleeding or hematoma formation, hypotension, tachycardia, weak peripheral pulses, decreased sensation and movement.
Continue to monitor your client closely, and prepare to assist with interventions to stop the bleeding. Now, moving on to client and family teaching.
General client and family teaching10:40–13:14
First, explain to your client mitral stenosis is a defect in the function of the mitral valve, a valve situated between their left atrium and left ventricle.
Let them know that because the valve is stiff, it doesn’t open properly, and this impairs the circulation of blood around the heart.
Explain that the balloon valvuloplasty opened the valve up to restore circulation. Then, review the plan of care, and encourage them to keep all follow up tests and appointments for continued monitoring and care.
Next, teach them how to care for themselves at home. Instruct them to keep the insertion site clean and dry.
Tell them they should avoid bathing for at least 5 days, and that they can shower with the insertion site covered. As far as activity, let them know they can take short walks, but that they should avoid vigorous exercise, bending, squatting, or lifting anything that weighs over 20 pounds or 9 Kg.
Stress the importance of monitoring the insertion site and to seek medical attention right away if they notice increased bleeding; signs of infection like swelling, redness, increased pain, fever, or chills; or if they experience numbness, tingling, or coolness of the affected extremity.
Also be sure to teach them to recognize signs that their repaired valve is not functioning properly, and to immediately seek emergency care if they experience dizziness, palpitations, chest pain, or trouble breathing.
Also talk to your client about lifestyle modifications that can help keep their heart healthy. Encourage them to maintain a healthy weight, and to engage in moderate physical exercise regularly, as prescribed by their healthcare provider.
Instruct them to eat plenty of fruits, vegetables, and whole grains, as well as lean meats, and low-fat or fat-free dairy products.
Provide them with a list of foods they can select from, and teach them how to choose foods that are low in sodium. Also review the importance of limiting their consumption of alcohol and caffeine, and to avoid any products that contain nicotine or tobacco, including cigarettes, e-cigarettes, and chewing tobacco.
Provide them with counseling and refer them to resources for support, if they need help quitting smoking. Also teach them about the importance of adequate sleep, review relaxation techniques, and talk to them about strategies they can use to deal with stressful situations.
Review13:14–14:40
Alright, as a quick recap. Valvular heart disease refers to damage or defect involving one or more heart valves.
The main cause of valvular heart disease is rheumatic fever following a strep throat infection, but other causes include infective endocarditis, atherosclerosis, myocardial infarction, and dilated cardiomyopathy.
Clients with valvular heart disease present with heart murmurs, as well as angina, fatigue, orthopnea, and dyspnea. Diagnosis is established by performing various tests such as electrocardiogram or ECG assessment, cardiac catheterization, a transthoracic or transesophageal echocardiogram or an exercise tolerance test.
Treatment of valvular heart disease consists of medical management, as well as surgical procedures that range from minimally invasive percutaneous procedures to open-heart surgeries in order to repair or replace the affected valves.
Nursing care for a client with mitral valve stenosis focuses on maintaining adequate cardiopulmonary function and monitoring for complications postoperatively.
General client and family education include promoting a healthy lifestyle, self-care at home, and when to seek medical care.
| VALVULAR HEART DISEASE | ||
| KEY POINTS | NOTES | |
| DEFINITION |
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| PHYSIOLOGY |
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| CAUSES AND RISK FACTORS |
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| PATHOPHYSIOLOGY |
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| SIGNS AND SYMPTOMS |
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| DIAGNOSIS |
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| TREATMENT |
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| MANAGEMENT OF CARE |
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| PATIENT AND FAMILY TEACHING |
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