Tubular reabsorption and secretion
Definitions & Key takeaways
Tubular reabsorption and secretion are important processes that occur in the kidneys to maintain the balance of electrolytes and fluids in the body. Tubular reabsorption occurs when the kidneys reabsorb useful substances, such as glucose, amino acids, and electrolytes, from the filtrate back into the bloodstream. This process occurs primarily in the proximal tubule of the nephron and is critical in maintaining the body's electrolyte and fluid balance. Tubular secretion is the opposite process, where the kidneys secrete waste products, such as excess potassium or hydrogen ions, from the bloodstream into the tubular fluid. This process occurs primarily in the distal and collecting tubules of the nephron.
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C4 (SA)
Renal system anatomy and physiology
Endocrine system anatomy and physiology
Glomerular filtration
Measuring renal plasma flow and renal blood flow
Regulation of renal blood flow
Renal clearance
Proximal convoluted tubule
Loop of Henle
Distal convoluted tubule
Tubular reabsorption and secretion
Tubular secretion of PAH
Tubular reabsorption of glucose
Urea recycling
Tubular reabsorption and secretion of weak acids and bases
Renin-angiotensin-aldosterone system
Diabetes mellitus
Syndrome of inappropriate antidiuretic hormone secretion (SIADH)
Diabetes insipidus
Insulin
Glucagon
Antidiuretic hormone
Movement of water between body compartments
Body fluid compartments
Sodium homeostasis
Diabetes mellitus: Clinical
Diabetes mellitus: Pathology review
Endocrine system: Diabetes mellitus
Hyponatremia
Hyponatremia: Clinical
Hypernatremia: Clinical
Hypernatremia
Diabetes insipidus and SIADH: Pathology review
Diabetic retinopathy
Gestational diabetes
Managing diabetes during the holidays: Information for patients and families
Insulins
Atherosclerosis and arteriosclerosis: Pathology review
Diabetic nephropathy
Diabetic ketoacidosis (DKA): Nursing process (ADPIE)
Hypertension
Hypertensive emergency
Lipid-lowering medications: Statins
Lipid-lowering medications: Fibrates
Miscellaneous lipid-lowering medications
Hypercholesterolemia: Clinical
Dyslipidemias: Pathology review
Hypertriglyceridemia
Familial hypercholesterolemia
Osmotic diuretics
Hypokalemia
Hypokalemia: Clinical
Hyperkalemia
Hyperkalemia: Clinical
Loop diuretics
Potassium sparing diuretics
Thiazide and thiazide-like diuretics
Hypoglycemics: Insulin secretagogues
Miscellaneous hypoglycemics
ACE inhibitors, ARBs and direct renin inhibitors
Carbonic anhydrase inhibitors
Thyroid nodules and thyroid cancer: Pathology review
Thyroid nodules and thyroid cancer: Clinical
Thyroid cancer
Hashimoto thyroiditis
Thyroid storm
Thyroid hormones
Hypothyroidism and thyroiditis: Clinical
Riedel thyroiditis
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Anatomy of the thyroid and parathyroid glands
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Parathyroid hormone
Hyperthyroidism
Graves disease
Toxic multinodular goiter
Hyperparathyroidism
Hypoparathyroidism
Hypercalcemia
Hypocalcemia
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Hyperthyroidism medications
Hypothyroidism medications
Osteogenesis imperfecta
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Acute tubular necrosis
Kidney stones: Clinical
Kidney stones
The role of the kidney in acid-base balance
Acid-base disturbances: Pathology review
Metabolic acidosis
Renal tubular acidosis
Respiratory acidosis
Renal tubular acidosis: Pathology review
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Potassium homeostasis
Plasma anion gap
Respiratory alkalosis
Metabolic alkalosis
Hydronephrosis
Renal artery stenosis
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Chronic kidney disease
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Membranous nephropathy
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Neurogenic bladder
Lower urinary tract infection
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Electrolyte disturbances: Pathology review
Renal failure: Pathology review
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Adrenocorticotropic hormone
Oxytocin and prolactin
Somatostatin
Synthesis of adrenocortical hormones
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Estrogen and progesterone
Phosphate, calcium and magnesium homeostasis
Congenital adrenal hyperplasia
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Hypothyroidism
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Hypopituitarism
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Adrenal masses: Pathology review
Cushing syndrome and Cushing disease: Pathology review
Multiple endocrine neoplasia: Pathology review
Adrenal hormone synthesis inhibitors
Mineralocorticoids and mineralocorticoid antagonists
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Parathyroid conditions and calcium imbalance: Clinical
MEN syndromes: Clinical
Glucocorticoids
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Metabolic and respiratory alkalosis: Clinical
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