Pressure injury: Nursing process (ADPIE)

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Pressure injury: Nursing process (ADPIE)

NSG1201

NSG1201

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Wound healing
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Peripheral arterial disease (PAD): Nursing process (ADPIE)
Pharmacodynamics: Nursing pharmacology
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Vital signs - Pain: Nursing skills
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Antacids: Nursing pharmacology
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Antibiotics - Trimethoprim and sulfonamides: Nursing pharmacology
Anticoagulants - Direct thrombin and factor Xa inhibitors: Nursing pharmacology
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Antiemetics: Nursing pharmacology
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Antihyperlipidemics - Statins: Nursing pharmacology
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Antiplatelet agents: Nursing pharmacology
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Antiretrovirals for HIV/AIDS - Integrase strand transfer inhibitors: Nursing pharmacology
Antiretrovirals for HIV/AIDS - NRTIs and NNRTIs: Nursing pharmacology
Antiretrovirals for HIV/AIDS - Protease inhibitors: Nursing pharmacology
Antivirals for herpesviruses: Nursing pharmacology
Antivirals for influenza: Nursing pharmacology
Anxiolytics and sedative-hypnotics: Nursing pharmacology
Beta-adrenergic blockers: Nursing pharmacology
Biologic agents: Nursing pharmacology
Blood products: Nursing pharmacology
Calcium-channel blockers: Nursing pharmacology
Cardiac glycosides: Nursing pharmacology
Chloramphenicol: Nursing pharmacology
Debridement agents: Nursing pharmacology
Direct-acting vasodilators: Nursing pharmacology
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Diuretics - Osmotic and carbonic anhydrase inhibitors: Nursing pharmacology
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Eye anesthetics: Nursing pharmacology
Gallstone-dissolving agents: Nursing pharmacology
Gastric mucosal protective agents: Nursing pharmacology
Glucocorticoids and mineralocorticoids: Nursing pharmacology
Hematopoietic growth factors: Nursing pharmacology
Hemostatics: Nursing pharmacology
Histamine H2 antagonists: Nursing pharmacology
Hormones and hormone modulators for cancer: Nursing pharmacology
Immunoglobulins: Nursing pharmacology
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Immunosuppressants for autoimmune diseases: Nursing pharmacology
Corticosteroids - Inhaled: Nursing pharmacology
Insulin: Nursing pharmacology
Iron preparations: Nursing pharmacology
Keratolytics: Nursing pharmacology
Laxatives: Nursing pharmacology
Leukotriene modifiers: Nursing pharmacology
Lung surfactants and antenatal corticosteroids: Nursing pharmacology
Medications affecting the parathyroid glands: Nursing pharmacology
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Mydriatics and cycloplegics: Nursing pharmacology
Nitrates: Nursing pharmacology
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Oral antidiabetic medications - Alpha-glucosidase inhibitors: Nursing pharmacology
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Pancreatic enzyme replacements: Nursing pharmacology
Pharmacokinetics - Distribution: Nursing pharmacology
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Plant extracts for chemotherapy: Nursing pharmacology
Platinum-based agents: Nursing pharmacology
Prostaglandins: Nursing pharmacology
Proton pump inhibitors (PPIs): Nursing pharmacology
Respiratory stimulants: Nursing pharmacology
Rho(D) immune globulin: Nursing pharmacology
Sympathomimetic medications: Nursing pharmacology
Thrombolytics: Nursing pharmacology
Tocolytics: Nursing pharmacology
Antibiotics - Topical: Nursing pharmacology
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Pressure injury: Nursing process (ADPIE)
Emergency care: Falls
Diabetic ketoacidosis (DKA): Nursing process (ADPIE)
Inflammatory process: Nursing
Nutrition - Oral: Nursing skills
Core measures: Nursing
Standards and methods of documentation: Nursing
Physical assessment - Abdomen: Nursing
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Vital signs - Blood pressure (BP): Nursing skills
Vital signs - Oxygen saturation (SpO2): Nursing skills
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Notes

PRESSURE INJURY

KEY POINTS
NOTES
PATIENT REPORT
  • 78-year-old
  • Residing in skilled nursing facility
  • History osteoarthritis and hip fracture
  • Redness and shallow ulcer on sacrum

PATHOPHYSIOLOGY
  • Caused by prolonged pressure on skin and underlying tissue 
  • Most common over bony prominences 
  • Pressure reduces blood flow
    • Tissue hypoxia
    • Ischemia
    • Necrosis
    • Ulceration 
  • Risk factors 
    • Immobility  
    • Advanced age 
    • Poor nutrition and hydration 
    • Prolonged exposure to skin irritants 
    • Conditions impairing blood flow  
  • Complications 
    • Pain  
    • Local wound infections 
    • Bacteremia
    • Sepsis
    • Death 
  • Staging 
    • Stage 1
      • Red, intact skin that does not blanch 
    • Stage 2
      • Partial-thickness skin loss, shallow open wound or blister 
    • Stage 3
      • Full-thickness tissue loss, may include slough 
    • Stage 4
      • Deep wound exposing muscle or bone, may include eschar 
    • Unstageable
      • Covered with slough or eschar, depth can't be determined

DIAGNOSIS AND TREATMENT
  • Diagnosis
    • Braden scale assessment
    • Nutritional assessments to evaluate healing potential
    • Laboratory tests
  • Treatment
    • General  
      • Redistribute pressure by regular repositioning  
      • Ensure adequate nutrition and hydration
      • Manage pain 
      • Monitor wounds 
        • Keep wounds clean, moist, and covered 
    • Stage 1 
      • Transparent or hydrocolloid dressing 
    • Stage 2 
      • Hydrocolloid or hydrogel dressing 
      • Alginate dressing for moderate to heavy exudate
    • Stage 3 and 4 
      • Hydrocolloid, hydrogel, foam, or alginate dressings 
      • Wet-to-dry gauze dressing
    • Stage 4 
      • May require surgical debridement
      • Negative pressure wound therapy 
    • Unstageable 
      • Surgical debridement 

ASSESSMENT
  • Vital signs 
    • Heart rate: 80 regular 
    • Respiratory rate: 18
    • Clear breath sounds bilaterally 
    • Blood pressure: 124/60 mmHg 
    • Temperature: 98.4°F (36.9°C) 
    • Pain: 2/10
  • Skin assessment 
    • Stage 2 pressure injury noted on sacrum 
    • Shallow open ulcer with reddish-pink wound bed 
    • No exudate or odor 
    • Measured and photographed for documentation 
  • Height: 5'5" (165 cm)
  • Weight: 110 lbs (49.9 kg)
  • BMI: 18.3 
  • Previous weight (1 month ago): 118 lbs (53.5 kg)
    • BMI: 19.6 
  • Reports decreased appetite and reduced enjoyment of food 
  • Most recent Braden scale score: 12

NURSING DIAGNOSES
  • Impaired skin integrity related to decreased mobility and presence of a stage 2 pressure injury
  • Imbalanced nutrition: less than body requirements related to insufficient dietary intake
  • Risk for infection related to open wound

PLANNING
  • By the end of the week
    • Patient's wound will show signs of healing
    • Patient will 
      • Ambulate with assistance
      • Change position regularly
      • Improve dietary intake 
  • Long-term goal
    • Wound remain free from infection

IMPLEMENTATION
  • Wound assessment performed every shift 
  • Notify HCP of signs or symptoms of infection
  • Certified nursing assistant (CNA) will
    • Assist with repositioning every hour 
    • Assist with ambulation every 2–4 hours
  • Collaborated with dietitian
  • Tailored meals to patient preferences
    • Protein
    • Vitamins
    • Minerals
  • Patient education
    • Discussed importance of
      • Increased activity to improve circulation
      • Healthy diet to support tissue repair 

EVALUATION
  • Dressing clean, dry, and intact 
  • Wound surface area has slightly decreased compared to initial measurement 
  • Patient reports assisted ambulation in the hallway 
  • Weight: 118.5 lbs (53.8 kg)

Transcript

Watch video only

Joann Mercer is a 78-year-old female client who resides in a skilled nursing facility.

Mrs. Mercer has a history of osteoarthritis and hip fracture.

She needs assistance to walk, and spends most of her time in bed or sitting in her wheelchair.

The certified nursing assistant, or CNA, who is taking care of Mrs. Mercer informs you of redness and a shallow ulcer that developed on her sacrum.

You are concerned that Mrs. Mercer has developed a pressure injury.

Pressure injuries, also known as decubitus ulcers, involve damage to the skin or underlying tissue that result from prolonged pressure.

Now, pressure injuries usually appear over bony prominences, especially the sacrum, followed by the heels, since these areas have the thinnest subcutaneous tissue between the bone and the skin.

So the prolonged pressure causes a reduced blood flow to that tissue area, resulting in tissue hypoxia and ischemia, and ultimately leading to necrosis and ulceration.

Most often, pressure injuries develop in clients who aren’t moving about, like those on chronic bedrest or consistently in a wheelchair.

Other factors that can increase the risk for skin injury are thinning of skin and subcutaneous tissue due to advanced age as well as dry skin and thin subcutaneous tissue due to inadequate nutrition and hydration; and prolonged contact to skin irritants like sweat, urine, and feces.

Other important risk factors for pressure injuries are conditions that may impair blood flow, such as heart and lung disease and diabetes mellitus.

Clients should be assessed for the risk of developing a pressure injury using a validated assessment tool like the Braden Scale.

This scale looks at six criteria, which include sensory perception, moisture, activity, mobility, nutrition, and friction or shear.

The lower the score, the higher the risk of injury.

Nutritional assessments can be used to assess the likelihood of injury as well as healing.

With non-healing injuries, laboratory tests can be done to assess an underlying cause like diabetes or infection.

Laboratory tests can include a glucose test, which would reveal hyperglycemia in case of diabetes, or a complete blood count showing elevated white blood cells, an elevated erythrocyte sedimentation rate, and elevated C reactive protein or CRP, as well as blood cultures to check for an infectious cause.

Now, pressure injuries can often cause symptoms like pain or pruritus, and can present with purulent drainage or bleeding.

In addition, pressure injuries can lead to complications like local infections of the wound.

In severe cases, the infection may spread and result in bacteremia, which can lead to sepsis, and death.

Pressure injuries are staged according to their level of tissue damage.

There are four stages of injury development and pain can present at any stage.

In stage 1 the skin will appear red, but remain intact.

When pressed, the area will not blanch or turn white.

At stage 2, there is partial-thickness skin loss, and the wound will look like a shallow open wound or blister.

Stage 3 involves full thickness loss of tissue that can present with slough or light-colored dead tissue.

In stage 4, the wound extends deep enough to expose muscle or bone.

Eschar, or dark-colored dead tissue, can be present.

Finally, if a pressure injury is completely covered with slough or eschar, it can be difficult to determine its depth; this is referred to as an unstageable pressure injury.

Treatment for pressure injuries depends on the stage of the wound, but generally involves redistribution of pressure, by regularly repositioning the client, as well as ensuring good nutrition and hydration to encourage wound healing; pain management; and frequent monitoring and wound care to keep the wound clean, moist, and covered.

For stage 1 injuries, either a transparent or hydrocolloid type dressing is used.

Both are useful in acting as a moisture barrier, as well as preventing shear and friction.

For stage 2 injuries, a hydrocolloid or hydrogel dressing can be used to assist with absorption, protection, and debridement of the wound.

An alginate dressing can be used if there is moderate to heavy exudate.

For stage 3 or 4 injuries, a hydrocolloid, hydrogel, foam, or alginate dressing can be used.