Prostatitis

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Prostatitis

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Development of the renal system
Ureter, bladder and urethra histology
Kidney histology
Renal system anatomy and physiology
Body fluid compartments
Hydration
Movement of water between body compartments
Horseshoe kidney
Renal agenesis
Potter sequence
Posterior urethral valves
Multicystic dysplastic kidney
Polycystic kidney disease
Vesicoureteral reflux
Alport syndrome
Urinary incontinence
Urinary incontinence: Pathology review
Neurogenic bladder
Bladder exstrophy
Antidiuretic hormone
Syndrome of inappropriate antidiuretic hormone secretion (SIADH)
Diabetes insipidus and SIADH: Pathology review
Diabetes insipidus
Nephrotic syndromes: Pathology review
Nephritic and nephrotic syndromes: Clinical
Nephritic syndromes: Pathology review
Minimal change disease
Hydronephrosis
Glomerular filtration
Measuring renal plasma flow and renal blood flow
Renal clearance
TF/Px ratio and TF/Pinulin
Regulation of renal blood flow
Sodium homeostasis
Kidney countercurrent multiplication
Urea recycling
Tubular reabsorption and secretion
Tubular reabsorption and secretion of weak acids and bases
Tubular secretion of PAH
Tubular reabsorption of glucose
Distal convoluted tubule
Loop of Henle
Proximal convoluted tubule
Renin-angiotensin-aldosterone system
Free water clearance
Amyloidosis
IgA nephropathy (NORD)
Poststreptococcal glomerulonephritis
Rapidly progressive glomerulonephritis
Lupus nephritis
Potassium homeostasis
Hypophosphatemia
Hyperphosphatemia
Hypermagnesemia
Hypomagnesemia
Hypocalcemia
Hypercalcemia
Hyperkalemia
Hypokalemia
Hyponatremia
Hypernatremia
Phosphate, calcium and magnesium homeostasis
The role of the kidney in acid-base balance
Acid-base disturbances: Pathology review
Physiologic pH and buffers
Renal tubular acidosis
Renal tubular acidosis: Pathology review
Metabolic acidosis
Metabolic and respiratory acidosis: Clinical
Respiratory acidosis
Metabolic alkalosis
Plasma anion gap
Respiratory alkalosis
Metabolic and respiratory alkalosis: Clinical
Acid-base map and compensatory mechanisms
Ornithine transcarbamylase deficiency
Kidney stones: Pathology review
Nitrogen and urea cycle
Goodpasture syndrome
Erythropoietin
Vitamin D
Kidney stones
ACE inhibitors, ARBs and direct renin inhibitors
Kidney stones: Clinical
Hypokalemia: Clinical
Renal tubular defects: Pathology review
Urinary tract infections: Clinical
Urinary tract infections: Pathology review
Lower urinary tract infection
Proteus mirabilis
Staphylococcus saprophyticus
Enterobacter
Klebsiella pneumoniae
Serratia marcescens
Pseudomonas aeruginosa
Renal artery stenosis
Thiazide and thiazide-like diuretics
Carbonic anhydrase inhibitors
Osmotic diuretics
Loop diuretics
Potassium sparing diuretics
Acute kidney injury: Clinical
Renal azotemia
Postrenal azotemia
Prerenal azotemia
Chronic kidney disease
Acute tubular necrosis
Renal papillary necrosis
Medullary cystic kidney disease
Chronic kidney disease: Clinical
Congenital renal disorders: Pathology review
Medullary sponge kidney
Chronic pyelonephritis
Acute pyelonephritis
Neisseria gonorrhoeae
Chlamydia trachomatis
Urethritis
Prostatitis
Schistosomes
Hemolytic-uremic syndrome
Thrombotic thrombocytopenic purpura
Renal cortical necrosis
Renal cell carcinoma
Angiomyolipoma
WAGR syndrome
Nephroblastoma (Wilms tumor)
Non-urothelial bladder cancers
Transitional cell carcinoma
Electrolyte disturbances: Pathology review
Renal failure: Pathology review
Renal and urinary tract masses: Pathology review
Transplant rejection
Graft-versus-host disease
Non-corticosteroid immunosuppressants and immunotherapies
Hypertension
BK virus (Hemorrhagic cystitis)

Key Takeaways

Prostatitis is inflammation of the prostate gland, which can be caused by a bacterial infection, though in many cases the cause is not understood. Prostatitis is classified into four major types. There is acute bacterial prostatitis which occurs when there is a sudden often severe infection of the prostate that's caused by bacteria. There is also chronic bacterial prostatitis characterized by a bacterial recurring or persistent infection. The third type is chronic pelvic pain syndrome characterized by chronic pain and discomfort in the pelvic area without signs of infection. Last but not least, there is asymptomatic inflammatory prostatitis, which is an inflammation of the prostate that typically presents with no symptoms. Treatment depends on the type and can include medications like antibiotics, anti-inflammatory drugs, and alpha-blockers.