Renal papillary necrosis
Renal papillary necrosis
Pathology
Acid-base physiology
Renal and ureteral disorders
Prerenal azotemia
Renal azotemia
Acute tubular necrosis
Postrenal azotemia
Renal agenesis
Horseshoe kidney
Potter sequence
Congenital renal disorders: Pathology review
Hyperphosphatemia
Hypophosphatemia
Hypernatremia
Hyponatremia
Hypermagnesemia
Hypomagnesemia
Hyperkalemia
Hypokalemia
Hypercalcemia
Hypocalcemia
Electrolyte disturbances: Pathology review
Hydronephrosis
Kidney stones
Kidney stones: Pathology review
Amyloidosis
Diabetic nephropathy
Focal segmental glomerulosclerosis (NORD)
Lupus nephritis
Membranoproliferative glomerulonephritis
Membranous nephropathy
Minimal change disease
Nephrotic syndromes: Pathology review
Alport syndrome
Goodpasture syndrome
IgA nephropathy (NORD)
Lupus nephritis
Poststreptococcal glomerulonephritis
Rapidly progressive glomerulonephritis
Nephritic syndromes: Pathology review
Acute pyelonephritis
Chronic pyelonephritis
Renal tubular acidosis
Renal tubular acidosis: Pathology review
Renal tubular defects: Pathology review
Renal papillary necrosis
Renal cortical necrosis
Polycystic kidney disease
Multicystic dysplastic kidney
Medullary cystic kidney disease
Medullary sponge kidney
Chronic kidney disease
Renal failure: Pathology review
Angiomyolipoma
Beckwith-Wiedemann syndrome
Nephroblastoma (Wilms tumor)
Renal cell carcinoma
WAGR syndrome
Renal and urinary tract masses: Pathology review
Renal artery stenosis
Bladder and urethral disorders
Key Takeaways
Renal papillary necrosis is a condition in which impaired blood supply to the kidneys leads to diffuse ischemia and subsequent necrosis of the renal papilla. Risk factors for renal papillary necrosis include diabetes, sickle cell anemia, and certain medications such as non-steroidal anti-inflammatory drugs (NSAIDs). Symptoms may include fever, flank pain, and blood in the urine. Treatment may include antibiotics, pain management, and in severe cases, surgery.