Anatomy clinical correlates: Thoracic wall

Last updated: November 01, 2022

Anatomy clinical correlates: Thoracic wall

pulmonary/resp

pulmonary/resp

Anatomy of the larynx and trachea
Bones and joints of the thoracic wall
Muscles of the thoracic wall
Vessels and nerves of the thoracic wall
Anatomy of the pleura
Anatomy of the lungs and tracheobronchial tree
Anatomy clinical correlates: Thoracic wall
Anatomy clinical correlates: Pleura and lungs
Development of the respiratory system
Nasal cavity and larynx histology
Trachea and bronchi histology
Bronchioles and alveoli histology
Respiratory system anatomy and physiology
Reading a chest X-ray
Lung volumes and capacities
Anatomic and physiologic dead space
Alveolar surface tension and surfactant
Compliance of lungs and chest wall
Combined pressure-volume curves for the lung and chest wall
Ventilation
Zones of pulmonary blood flow
Regulation of pulmonary blood flow
Pulmonary shunts
Ventilation-perfusion ratios and V/Q mismatch
Breathing cycle
Airflow, pressure, and resistance
Ideal (general) gas law
Boyle's law
Dalton's law
Henry's law
Graham's law
Gas exchange in the lungs, blood and tissues
Diffusion-limited and perfusion-limited gas exchange
Alveolar gas equation
Oxygen binding capacity and oxygen content
Oxygen-hemoglobin dissociation curve
Carbon dioxide transport in blood
Breathing control
Pulmonary chemoreceptors and mechanoreceptors
Pulmonary changes at high altitude and altitude sickness
Pulmonary changes during exercise
Choanal atresia
Laryngomalacia
Allergic rhinitis
Nasal polyps
Upper respiratory tract infection
Sinusitis
Laryngitis
Retropharyngeal and peritonsillar abscesses
Bacterial epiglottitis
Nasopharyngeal carcinoma
Tracheoesophageal fistula
Congenital pulmonary airway malformation
Pulmonary hypoplasia
Neonatal respiratory distress syndrome
Transient tachypnea of the newborn
Meconium aspiration syndrome
Apnea of prematurity
Sudden infant death syndrome
Acute respiratory distress syndrome
Respiratory distress syndrome: Pathology review
Decompression sickness
Cyanide poisoning
Methemoglobinemia
Emphysema
Chronic bronchitis
Asthma
Cystic fibrosis
Bronchiectasis
Alpha 1-antitrypsin deficiency
Restrictive lung diseases
Sarcoidosis
Idiopathic pulmonary fibrosis
Pneumonia
Pneumonia: Pathology review
Klebsiella pneumoniae
Legionella pneumophila (Legionnaires disease and Pontiac fever)
Croup
Bacterial tracheitis
Lung cancer and mesothelioma: Pathology review
Lung cancer
Mesothelioma
Pancoast tumor
Superior vena cava syndrome
Pleural effusion, pneumothorax, hemothorax and atelectasis: Pathology review
Pneumothorax
Pleural effusion
Pulmonary edema
Pulmonary hypertension
Pulmonary embolism
Deep vein thrombosis and pulmonary embolism: Pathology review
Cystic fibrosis: Pathology review
Mycobacterium tuberculosis (Tuberculosis)
Tuberculosis: Pathology review
Obstructive lung diseases: Pathology review
Restrictive lung diseases: Pathology review
Apnea, hypoventilation and pulmonary hypertension: Pathology review
Sleep apnea
Antihistamines for allergies
Bronchodilators: Beta 2-agonists and muscarinic antagonists
Bronchodilators: Leukotriene antagonists and methylxanthines

Transcript

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If it wasn't for our thoracic wall, many of the important organs in our thoracic cavity would be unprotected and vulnerable to injury.

But sometimes the thoracic wall itself can be injured, which has a wide range of clinical consequences depending on the affected structures.

Let’s start by talking about the ribs.

First, like any other bone in our body, ribs can break, causing rib fractures.

These usually result from direct trauma or crushing injuries.

The middle ribs are the ones most commonly fractured.

The weakest part of a rib is the posterolateral bend, anterior to its angle.

However, direct trauma can cause a rib to fracture anywhere.

The broken part of the rib can harm internal organs, such as the liver, kidney or the spleen.

Rib fractures higher up can cause mediastinal injuries, and if the fracture is lower, then it can tear the diaphragm.

Furthermore, rib fractures at any level have the risk of causing an intrathoracic injury such as a pneumothorax, which is when there’s air in the pleural cavity, and that doesn’t allow the lung on that side to expand properly.

Since ribs move during respiration, coughing, laughing and sneezing are very painful after a rib fracture!

A related injury is a flail chest, which is when three or more ribs fracture in two or more places, which can allow a big segment of the thoracic wall to move freely.

During a normal inspiration, the thoracic wall expands outwards and increases its diameter, whereas during expiration, it decreases its diameter to expel air.

However, when there’s a flail chest, the movement is paradoxical, meaning that during inspiration, the free segment actually moves inward and during expiration, it moves outward.

This is an extremely painful injury that impairs ventilation, and, as a consequence, blood isn’t properly oxygenated.

Management wise, for a flail chest, you want to ensure adequate pain control and supplemental oxygen if needed.

If respiratory failure occurs as a result of the flail chest, then positive pressure ventilation can be used to force the flail chest segment out during inspiration.

Sometimes, a chest tube may also be required.

Now, between the ribs, in the anterior part of the chest, there’s the sternum, which is the protector of the mediastinal viscera.

Sources

  1. "Hyman's Comparative Vertebrate Anatomy" University of Chicago Press (1992)
  2. "Anatomy & Physiology" Wikipedia (2009)
  3. "Congenital Thoracic Wall Deformities" Springer Science & Business Media (2011)
  4. "Median sternotomy" Multimedia Manual of Cardio-Thoracic Surgery (2015)
  5. "Management of Congenital Chest Wall Deformities" Seminars in Plastic Surgery (2011)
  6. "Mosby's Medical Dictionary" Elsevier (2013)